For several samples, we observed an over-expression of exon 7 and an under-expression of exon 8 of AKT3, and an over-expression of exon 4 and alternative exon 6 donor splice site for VEGFA. Although the relationships between SRSF2 status and these splicing events were less clear in these cases, maybe owing to the small number of samples, these data validated, in cancer samples, some of the pre-mRNA splicing events detected in the SRSF2-over-expressing H538 cell line. The gene discussed is SRSF2; the disease is cancer.