Type A thymomas were enriched for three pathways: among the “cell migration genes,” over-expressed KAl1 has anti-metastatic function (30); IL8RB reinforces p53-dependent senescence (31); and SAA1 expression that has not been noted in thymomas before, can have pro- or anti-apoptotic functions in context-dependent manner (32–36). Here, TP53 is linked to thymoma.