The common clinical features of this condition include liver cirrhosis secondary to viral hepatitis or other factors, high serum levels of AFP [2] and CA19-9 (in our case, the CA19-9 level was high but the AFP level was normal), history of cholangitis with dilation of the intra- and extrahepatic bile ducts, rapid development of liver dysfunction with jaundice, and a filling defect in the common bile duct of 2 cm or longer. The gene discussed is AFP; the disease is animal viral hepatitis.