FOXP1 and B-cell neoplasm: Collectively, we provide evidence that FOXP1 is the target of two molecularly distinct types of rearrangements in B-cell neoplasms: (i) t(3;14)(p13;q32)/IGH-FOXP1, which dysregulates expression of FOXP1FL, and (ii) non-IG aberrations, which result in ectopic expression of FOXP1NT.