IFI30 and autoimmune disease: As many other autoantigens contain disulfide bonds, e.g., TRP2, gp100, types VII and XVII collagen, insulin, glutamic acid decarboxylase, and thyroid-stimulating hormone receptor (5, 42–49), GILT is likely to alter the MHC class II-restricted epitopes that are presented and the pathogenesis of other autoimmune diseases.