As reviewed by Paydas [31], in lymphomas and nasopharyngeal carcinomas, tumour cell infection with EBV is related to increased angiogenesis, in particular because the viral late membrane protein 1 (LMP1) induces expression of VEGF and activation of PTGS2, interleukin 8 (IL8), fibroblast growth factor 2 (FGF2) and other pro-angiogenic factors. Here, FGF2 is linked to lymphoma.