Zinc, as a micronutrient, plays an important role in the regulation of the T cell–mediated function (14-16); Even though, zinc deficiency has been shown to cause thymus involution and to depress lymphocyte proliferation, interleukin-2 (IL-2) production, delayed-type hypersensitivity skin responses, and antibody response to T cell–dependent antigens (14, 17). This evidence concerns the gene IL2 and Zinc deficiency.