SNAI2 and benign prostatic hyperplasia: In an unrelated study, the invasive tumorigenic clones derived from the benign prostatic hyperplasia (BPH)-1 cell line chronically exposed to TGF-β display prominent EMT features with high levels of SNAI2, ZEB1, and ZEB2, all confirmed miR-200 targets; however, the basal miR-200 levels in these cells remain unaltered suggesting a divergent mechanism [34].