However, in a separate study [81], the influence of host cell gene transcription on proviral latency was analyzed and compared for five different models of latency including the Siliciano [17] and Planelles [15] models, a Jurkat model with polyclonal integration [78], infection of primary resting CD4+ T cells [82], and infection of primary activated CD4+ T cells [82]. Here, CD4 is linked to infection.