To further characterize the function of miR-26a in vivo and in vitro, they utilized an adenoassociated virus carrying miRNA-26a gene to infect HepG2 cells and mouse model with MYC-induced hepatocarcinoma and found out that ectopic expression of miR-26a results in blockage of cell proliferation and induction of tumor cell apoptosis, suggesting that delivery of miRNAs with tumor suppressive function may provide a novel strategy to develop miRNA therapy. The gene discussed is MYC; the disease is neoplasm.