On the contrary, in those tumors where theBax gene is less expressed, a higher level ofBcl-2, resulting in a prevalence of Bcl-2 complexes,would be able to block the apoptoticpathways, thus causing cancer progression.This fact contrasts with the hypothesis thatthe relative ratio between Bcl-2 and Bax proteinshas the capability of controlling the susceptibilityof cells to death stimuli. Here, BCL2 is linked to cancer.