DMD and Duchenne muscular dystrophy: However, the miR-206 control of KSRP expression that we document in the current work promotes an increase in the endogenous expression of utrophin A. This is important, as utrophin A upregulation is currently envisaged as a potential therapy for DMD because it can functionally compensate for the absence of dystrophin along the sarcolemma of dystrophic muscle fibers.