This proliferation was specific to LILRA3 because similar experiments performed with glutamate decarboxylase-65 (GAD-65), which is an autoantigen in type-1 diabetes but should be an immunologically irrelevant protein in healthy donors, did not induce a proliferation in CFSE-stained leukocytes (Figure S1A). The gene discussed is GAD2; the disease is type 1 diabetes mellitus.