In agreement with this pathogenic process, serum HDL-c has been previously demonstrated as negatively correlated with serum LCN2 levels [18]; likewise, in the current study serum TG was identified as a positive influencing factor of serum LCN2 levels, indicating that the related pathogenic mechanism of atherosclerosis may involve disruption of lipid metabolism. Here, LCN2 is linked to atherosclerosis.