ALDH1A2 and Dysphagia: Selective rescue of LgDel CN V phenotypes by heterozygous mutation of Raldh2 (Raldh2+/−), which lowers RA signaling in the whole embryo by 20% (Maynard et al., 2013), establishes RA-mediated disruption of CN development as a potential contributor to dysphagia pathogenesis.