Most of them remain relatively small and rarely enlarge, become symptomatic, or rupture.10 In a consecutively arteriography follow-up of SAAs secondary to liver cirrhosis in six patients it was found that the long diameter of the SAAs only increased by 0.18 mm on average.11 Various therapeutic options for SAAs are available which include endovascular management, laparoscopic surgery, and open surgery, although their indications and applications as standard therapy remain controversial. This evidence concerns the gene PCSK1N and cirrhosis of liver.