Considering the shared genetic component among the different immune-mediated diseases, including the autoimmune uveitis [12,13,28], and the key role that Th17 responses play in the uveitis pathogenesis, we speculated that STAT4 and IL23R, two general autoimmunity loci, may contribute to the non-anterior uveitis susceptibility. This evidence concerns the gene IL23R and autoimmune uveitis.