Experimentally, it has been proven that molecular mimicry is only able to trigger autoimmune diabetes provided that there is a full identity between the amino acid sequences as modeled in the RIP-gp (rat insulin promoter) mice which expresses a glycoprotein (gp) of the lymphocytic choriomeningitis virus (LCMV) in their β-cells and after an infection rapidly develop diabetes [24, 103, 104]. This evidence concerns the gene INS and diabetes mellitus.