Tyrosine kinase inhibitors, through targeting tyrosine kinase domains of platelet-derived growth factor receptor α/β, vascular endothelial growth factor receptors 1, 2, and 3, mitogen-activated protein kinase (MAPK)/extracellular signal-regulated kinases (ERK), and v-raf murine sarcoma viral oncogene homolog B1 (BRAF) inhibit the proliferation, migration, differentiation, and survival of cancer cells [5–9]. This evidence concerns the gene BRAF and cancer.