ADRB2 and ovarian carcinoma: A recent study suggested yet another pathway via which stimulation of the β2-AR can promote tumor growth: activation of FAK, an intra-cellular factor that is associated with protection of ovarian cancer cells from anoikis (death after separation from extracellular matrix), increased survival time of detached cells, facilitated their migration, and promoted their reattachment and colonization in secondary sites [70].