Given that elevated inflammation due to TNF-α, IL-6 and IL-1β are amongst the causative factors of liver fibrosis and liver steatosis [10], and high levels of TNF-α, IL-6 and IL-1β are known to deregulate glucose metabolism [14], [49], we measured the levels of these cytokines in the liver. The gene discussed is TNF; the disease is Hepatic steatosis.