ATP13A2 and Lewy body dementia: Double-immunolabeling of ATP13A2 with β-amyloid in all cases confirmed that diffuse β-amyloid plaques were present in the DLB but not the PD or control cases, but revealed no colocalization or even any physical association between ATP13A2-positive neurons and β-amyloid-immunopositive plaques (Figure 1E).