For example, integration of lung expression and asthma genetic association data led to the identification of GSDMA as the most likely causal gene for asthma in the 17q21 region [Hao et al., 2012], despite a graphical representation of the data showing that the SNPs most strongly associated with GSDMA expression were relatively weakly associated with asthma, and, vice versa, that the SNPs most strongly associated with asthma showed relatively weaker levels of association with GSDMA expression compared to the strongest signals. This evidence concerns the gene GSDMA and asthma.