Whilst there were no genes identified either up or downstream from significant SNPs, that could have been directly implicated with a fatness phenotype, other significant SNPs that resided within genes included ALGA0039880 in the gene TINAG (tubulointerstitial nephritis antigen), INRA0024695 and ALGA0040094 both in the gene DST (dystonin) and DIAS0000554 in the gene GLO1 (glyoxalase I). The gene discussed is TINAG; the disease is nephritis.