It is not uncommon for nonmyeloid malignancies such as T-cell lymphomas [3] and Hodgkin's disease [4] to be associated with secondary eosinophilia due to the production of cytokines such as IL-3, IL-5, and GM-CSF which promote eosinophil differentiation and survival. This evidence concerns the gene IL3 and T-cell non-Hodgkin lymphoma.