Given that the molecular and cellular mechanisms underlying the MCPIP1 deficiency-induced anemia and immune disturbance in the developmental stages are not clear, we will in the future use the bone marrow MCPIP1−/− chimeric mice as a valuable animal model for this purpose by monitoring the hematopoietic cell differentiation and maturation over time following the bone marrow transplantation. This evidence concerns the gene ZC3H12A and anemia.