Its levels correlate with complications following myocardial infarction, and anti-IL-8 antibodies prevented injury in experimental models.7 Its levels also correlated well with left ventricular wall motion abnormalities in the postoperative period.4 Furthermore, it was reported that cyclic alveolar stress due to mechanical ventilation led to increased IL-8 levels.14 Here, CXCL8 is linked to myocardial infarction.