Under chemotherapy, cancer cells can gradually develop drug resistance that is acquired, for instance, by overexpression of transporter proteins (e.g., those belonging to the ATP-binding cassette type) [2,3] and fractionation of the cancerous stem cells [4] (which are less sensitive to exposure to cytostatics than more differentiated cancer cells), plus AKT [5,6] and NF-kappaB [7,8] overexpression as a compensatory response to administered cytotoxic drugs. This evidence concerns the gene AKT1 and cancer.