In contrast, lymphocytes from oxt-KO mice eating L. reuteri imparted delayed wound repair processes (Figs. 7a and 7b) with delayed collagen deposition (Figs. 7c) and increased inflammatory component (Fig. 7d) resembling aspects of neoplasia-associated inflammation [52]; indeed, other studies show CD4+CD45RBloCD25+ Tregs from immune-competent donor animals were similarly sufficient to inhibit inflammation-associated carcinogenesis in certain tissues [39]. The gene discussed is CD4; the disease is neoplasm.