To evaluate the contribution of a functional OXPHOS to HIV-GFP infection in ρ+ HOS cells we used three mitochondrial poisons to block respiration at the level of complex III (antimycin A), to inhibit the functioning of the F1FO-ATPase (oligomycin), or to dissipate the proton gradient across the mitochondrial inner membrane, required to drive the synthesis of ATP (the uncoupler CCCP). The gene discussed is DNAH8; the disease is infection.