For instance, in the focal adhesion pathway, the MYL2 and SPP1 interacted in the control (CC = 0.86), but they were not correlated with each other in the pmAF patients (CC = 0.17); although all of the connections among the DEGs in the dilated cardiomyopathy pathway were weak correlation in both pmAF patients and controls, there are great difference between the corresponding CCs in both cases. This evidence concerns the gene MYL2 and dilated cardiomyopathy.