In Tukey–Kramer post hoc test, we found KAL1 was significantly upregulated in tumors of glia/astrocyte origin (e.g., glioblastoma and astrocytoma), compared with tumors of non-glia (e.g., meningioma) or non-brain origin (e.g., adenocarcinoma). This evidence concerns the gene ANOS1 and astrocytoma (excluding glioblastoma).