Human infants with BPD have low levels of CCSP, SCGB3A1, and SCGB3A2 gene expression in the surface epithelium of the upper and lower airways suggesting that injury and/or remodeling of airway epithelium that express SCGB family proteins is a pathologic feature of BPD [10]. This evidence concerns the gene SCGB3A2 and bronchopulmonary dysplasia.