Of note, poor acetylation of amino acids and proteins at birth will be a risk factor for developing T1D and other autoimmune diseases, such as autoimmune thyroid disease56 and CD57 (CD was found primarily associated with T1D in our cohort), but it must be pointed out that the ‘risk' applied only in HLA genetically susceptible individuals (for example, HLA DR3/DR4) and not in individuals with other HLA haplotypes. The gene discussed is B3GAT1; the disease is type 1 diabetes mellitus.