Additional observations of SLC1A1 deletions in disease cohorts include one exonic deletion in 235 subjects with both SCZ and epilepsy (32), a single exonic deletion in 459 unrelated adults with schizophrenia (33), one exonic deletion among 1637 German patients with schizophrenia or schizoaffective disorder (34) and Cooper et al. (17) report an enrichment of SLC1A1 deletions in neurological, craniofacial and epilepsy cases. This evidence concerns the gene SLC1A1 and epilepsy.