However, our findings showing that the extra copies of the ALK gene observed in 64% of the IBC patients were largely associated with aneusomy of chromosome 2 led us to a different conclusion than that of Robertson et al. The results of our comprehensive evaluation of the status of ALK gene does not support the presence of EML4-ALK gene rearrangements in IBC. The gene discussed is ALK; the disease is inflammatory breast carcinoma.