The observations that KGF was dramatically up-regulated following cutaneous injury in mouse and human full-thickness wounds [30, 31] as well as after tissue damage in the models of surgical bladder injury [26], chemically induced kidney injury [29], exposure of neonatal lungs to hyperoxia [27], and acute lung injury resulting from bleomycin injection [25], suggested that KGF participated in a wide variety of epithelial preservation and/or repair processes. Here, FGF7 is linked to kidney injury.