FOXM1 and hereditary hemorrhagic telangiectasia: HHT upregulated the level of mature miR-370 time- and dose-dependently, and anti-miR-370 treatment reversed HHT-induced apoptosis, so the miR-370–FoxM1 pathway might be a new mechanism for HHT-induced apoptosis with a positive feedback loop between miR-370 and HHT.