Epstein-Barr virus is the human γHV most commonly associated with the development of lung fibrosis[4,6,7], while in mice the normally non-pathogenic γ-HV MHV 68 induces pulmonary fibrosis in Th2-biased IFNγR-/- mice[8,9]. The gene discussed is IFNGR1; the disease is pulmonary fibrosis.