In the inguinal AT, Aa infection induced a significantly higher proportion of SFAs (Aa group: p = 0.027; Cpn + Aa group: p = 0.009; Figure 2A) and a lower proportion of PUFAs (Aa group: p = 0.006; Cpn + Aa group: p = 0.022; Figure 2C) compared to the control mice. This evidence concerns the gene CPN1 and infection.