The main two arguments in favor of an anti-c-MYC therapy are that (i) tumors are usually addicted to c-MYC and that even short-term interruption of c-MYC expression may drive tumor cells into apoptosis, rendering sustained anti-c-MYC therapy unnecessary [13], and (ii) that most normal cells are quiescent and side effects of c-MYC inhibiting proliferation of normal cells in the skin, the intestine and the hematopoietic system are relatively weak and reversible, and may be well tolerated [11]. The gene discussed is MYC; the disease is neoplasm.