However, since the ‘De Ritis Ratio’ of AST:ALT, proposed to distinguish between NAFLD and AFLD in the case of elevated levels of liver enzymes [62], among those 29 FLD patients lacking information on alcohol use in our study was <1 in the case of elevated ALT, AST and GGT levels, excessive alcohol intake turned out to be an unlikely cause of FLD in these patients. This evidence concerns the gene GPT and metabolic dysfunction-associated steatotic liver disease.