PDLIM7 and disease arising from reactivation of latent virus: However, it is interesting to note that parallels with other herpes viruses may be apparent: CD4+ T cells specific for a latent protein of EBV (LMP1) are able to secrete the immunosuppressive cytokine cIL-10 which is thought to play a role in evading immune responses during latent infection and maintenance of EBV latency [37]–[39].