Furthermore, this study could plug the crucial deficit in establishing gradient incidence of EGFR mutation across ethnicities and help inform designing of genome wide association studies to determine haplotype that may confer differential susceptibility to somatic mutations in EGFR in NSCLC, as reported for somatic mutations in JAK2 in human myeloproliferative neoplasms [37]. The gene discussed is EGFR; the disease is myeloproliferative disorder.