The application of loglinear analysis for these data allowed us to infer that there was not an interaction between the two polymorphisms for susceptibility to T2DM (χ2 = 1.04, DF = 1, p = 0.309) or an isolated effect of GSTM1 (χ2 = 1.097, DF = 2, p = 0.578). This evidence concerns the gene GSTM1 and type 2 diabetes mellitus.