In agreement with these findings recent analysis of prostate cancer animal models revealed that elevations of plasma cholesterol cause accumulations of cholesterol in lipid rafts and consequently leads to reduced apoptosis and increased tumor growth via Akt signaling (Li and Park, 2006; Adam et al., 2007; Llaverias et al., 2011; Pelton et al., 2012). This evidence concerns the gene AKT1 and neoplasm.