IL17A and allergic asthma: Because neutralization of IL-17A at antigen challenge does not protect mice against the development of AHR (Figure 1), and the potentiation of IL-17A and IL-17F production in the setting of a diminished Th2 response is not sufficient to exacerbate AHR, we hypothesized that the Th17 response contributes minimally to the airway inflammation and AHR development in our allergic asthma model.