SOD1 and Duchenne muscular dystrophy: This is compatible with previous evidence that neuromuscular junctions from symptomatic SOD1(G93A) EDL muscle display post-synaptic fragmentation and decreased acetylcholine receptors density ( [46]; but see 41), and similarly with what has been reported to occur during aging [49] and in neuromuscular junction disorders such as Duchenne Muscular Dystrophy [50].