Participation of TEP1 and FBN9 as final effectors of the JNK antiplasmodial response was explored by co-silencing these genes with puc. Co-silencing TEP1 increased the median number of oocysts/midgut from 1 to 21.5 (p<0.0001; KS test) and the prevalence of infection from 53% to 88% (p<0.02, χ2 test) relative to silencing puc alone (Figure 3C, Table S3). Here, TEP1 is linked to infection.