Studies have shown that over-expression of inflammatory cytokines considered responsible for the disorders observed during the course of the disease [9], and the rapid production of IFN-α/β are important for antiviral and autoimmune responses, leading to induced expression of hundreds of interferon-stimulated genes (ISGs) whose products direct antiviral and immunomodulatory actions that can defense against viral infections [10,11]. The gene discussed is IFNA1; the disease is viral infectious disease.